ศูนย์นวัตกรรมเทคโนโลยีหลังการเก็บเกี่ยว

Blossom-end rot of pears caused by Botrytis cinerea.

Kock, L. de; Combrink, J. C.;

Acta Horticulturae Year: 1994 Issue: No. 368 Pages: 667-672 Ref: 7 ref.

1994

บทคัดย่อ

ABSTRACT

A histological study was made of the systemic growth of B. cinerea from styles, stamens and sepals to the flower receptacle and mesocarp of immature pear fruit. At c. 75% petal fall (7 d after full bloom), styles started to dieback gradually due to natural senescence. Filaments, however, remained partly green until harvest. In most styles, hyphal growth ceased after full blossom. Hyphae never passed through styles into the carpel. Unlike the styles, hyphae in filaments grew without restriction and progressed, via vascular tissue, through sepals into tissues of the upper end of the flower receptacle, or of the mesocarp adjoining the sepals, without causing symptoms. B. cinerea entered filaments and spread into the receptacle or mesocarp at any time between blossoming and harvest and then became latent in these tissues. It is concluded that infected stamens are important sites of latent infection in pear. Filaments were more susceptible at the following stage. After 2 months, floral tubes closed,

and the stamens were protected from infection. It is suggested that control of blossom-end rot could be achieved if pears are sprayed with fungicide at 75-100% petal fall (when most stamens are exposed) and a month later (before floral tubes start to close).